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Originally posted by 6IXSTRINGJACK:
Go fuck yourself if you're trying to tell everyone that you don't have any bad and unhealthy habits.

I enjoy smoking. I don't particularly enjoy the idea of injecting a Chinese/Russian/US collaboration approved by Bill Gates and the CDC into my veins.

I know exactly what I'm inhaling.

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SIX: I don't particularly enjoy the idea of injecting a Chinese/Russian/US collaboration approved by Bill Gates and the CDC into my veins.

Do Right, Be Right. :)

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Originally posted by 1KIKI:
By sheer numbers, Portland doesn't come close to the 400,000+ person Sturgis superspreader event, which was followed by sharp outbreaks in both N (150/M -> 350/M) and S (100/M -> 400/M) Dakota.

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Originally posted by captaincrunch:

You got me on the bad habit though - my worst habit is I reply to your stupid sh*t.

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Originally posted by SIGNYM:

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SIX: I don't particularly enjoy the idea of injecting a Chinese/Russian/US collaboration approved by Bill Gates and the CDC into my veins.

Do Right, Be Right. :)

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If you really think that Russia and the USA are collaborating on anything then you need to dial back your conspiracy theories by... a whole lot.

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Originally posted by 6IXSTRINGJACK:

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Originally posted by captaincrunch:

You got me on the bad habit though - my worst habit is I reply to your stupid sh*t.

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Who knows...

Maybe one day it might kill you.

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SIX: I don't particularly enjoy the idea of injecting a Chinese/Russian/US collaboration approved by Bill Gates and the CDC into my veins.

SIGNY: If you really think that Russia and the USA are collaborating on anything then you need to dial back your conspiracy theories by... a whole lot.

SIX: Why is that?

So what that Trump and Putin seem to get along fine. There's nothing wrong with the US and Russia getting along.

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Especially when China is a much greater threat that our legacy media just finds it impossible to say anything negative about.

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Overpopulation is the world's biggest problem today, which is also something that nobody ever wants to talk about either. It is also a problem that effects every single person on the planet, no matter their race, sex, nationality, religion or the political makeup of their country.

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Trust me, there are people with a lot of money and power that talk about it every day. And there is zero doubt that controlling said population is one of the most important agendas on all of their dockets.

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Originally posted by 1KIKI:
Maybe you could dig thru the internet and come up with more solid numbers, and pursue this on your own.

But even if there were 40k professional and dedicated amateur rioters 'roaming' through the US - and I seriously doubt it's even 1k - your assumed 40k is still only a tenth of Sturgis.

And you have to remember this about Sturgis, people make a point of coming from all over the country to get together there and mingle in the same place and at the same time ... and then going back home.

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Originally posted by 1KIKI:
of note for Signy and me


The coronavirus may have reached Los Angeles even before China announced its outbreak

Researchers from UCLA and their colleagues at the University of Washington documented an unmistakable uptick in patients seeking treatment for coughs. The increase began the week of Dec. 22, 2019, and persisted through the end of February.

The researchers didn’t conduct any diagnostic tests, so they can’t say with certainty when doctors first encountered anyone infected with the virus that came to be known as SARS-CoV-2. But if the coronavirus had indeed been spreading under the radar since around Christmas, the pattern of patient visits to UCLA facilities would have looked a lot like what actually happened, they wrote in a study published Thursday in the Journal of Medical Internet Research.

https://news.yahoo.com/coronavirus-may-reached-los-angeles-144529852.h
tml

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As wearing a mask may reduce the amount of virus to which a wearer is exposed, it has been postulated that even if a mask-wearing person becomes infected, the reduced viral load to which they were exposed may mean that they suffer a milder disease. Supporting this theory is a study that showed that hamsters protected with a surgical mask partition were less likely to become infected with SARS-CoV-2 than those without the partition, and those that did get sick had milder illness. In addition, on cruise ships with COVID-19 outbreaks, the majority of infected patients (81%) were asymptomatic on a ship that had provided masks to all passengers and staff compared to only 18% of cases being asymptomatic on a cruise ship without masking (Gandhi, J Gen Intern Med 2020; Gandhi, N Engl J Med 2020).

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https://www.consumerlab.com/answers/how-to-make-covid-19-mask-at-home-
as-effective-as-n95

This is behind a pay wall, you may not be able to access the site.

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Pity would be no more,
If we did not MAKE men poor - William Blake

#WEARAMASK

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Originally posted by JEWELSTAITEFAN:
Proof that Covid was created in Wuhan Lab?


https://ca.news.yahoo.com/chinese-virologist-claims-she-proof-02570016
4.html

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Originally posted by 1KIKI:
By sheer numbers, Portland doesn't come close to the 400,000+ person Sturgis superspreader event, which was followed by sharp outbreaks in both N (150/M -> 350/M) and S (100/M -> 400/M) Dakota.

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Originally posted by 1KIKI:
COVID-19 Can Wreck Your Heart, Even if You Haven’t Had Any Symptoms

A growing body of research is raising concerns about the cardiac consequences of the coronavirus


We’ve also known for a while that some COVID-19 patients’ hearts are taking a beating, too—but over the past few weeks, the evidence has strengthened that cardiac damage can happen even among people who have never displayed symptoms of coronavirus infection. And these frightening findings help explain why college and professional sports leagues are proceeding with special caution as they make decisions about whether or not to play.

More than a dozen athletes at Power Five conference schools have been identified as having myocardial injury following coronavirus infection, according to ESPN; two of the conferences—the Big Ten and the Pac-12—already have announced they are postponing all competitive sports until 2021. And in Major League Baseball, Boston Red Sox ace pitcher Eduardo Rodriguez told reporters that he felt “100 years old” as a result of his bout with COVID, and of MLB’s shortened season because of myocarditis—an inflammation of the heart muscle, often triggered by a virus. Said Rodriguez: “That’s [the heart is] the most important part of your body, so when you hear that … I was kind of scared a little. Now that I know what it is, it’s still scary.”

Why are these athletes (and their leagues and conferences) taking such extreme precautions? It’s because of the stakes. Though it often resolves without incident, myocarditis can lead to severe complications such as abnormal heart rhythms, chronic heart failure and even sudden death.

Here’s the background: Myocarditis appears to result from the direct infection of the virus attacking the heart, or possibly as a consequence of the inflammation triggered by the body’s overly aggressive immune response. And it is not age-specific: In The Lancet, doctors recently reported on an 11-year-old child with multisystem inflammatory syndrome (MIS-C)—a rare illness—who died of myocarditis and heart failure. At autopsy, pathologists were able to identify coronavirus particles present in the child’s cardiac tissue, helping to explain the virus’ direct involvement in her death. In fact, researchers are reporting the presence of viral protein in the actual heart muscle, of six deceased patients. Of note is the fact that these patients were documented to have died of lung failure, having had neither clinical signs of heart involvement, nor a prior history of cardiac disease.

Ossama Samuel, associate chief of cardiology at Mount Sinai Beth Israel in New York, told me about a cluster of younger adults developing myocarditis, some of them a month or so after they had recovered from COVID-19. One patient, who developed myocarditis four weeks after believing he had recovered from the virus, responded to a course of steroid treatment only to develop a recurrence in the form of pericarditis (an inflammation of the sac surrounding the heart). A second patient, in her 40s, now has reduced heart function from myocarditis, and a third—an athletic man in his 40s—is experiencing recurring and dangerous ventricular heart rhythms, necessitating that he wear a LifeVest defibrillator for protection. His MRI also demonstrates fibrosis and scarring of his heart muscle, which may be permanent, and he may ultimately require placement of a permanent defibrillator.

J.N. ... told me that COVID-19 symptoms first appeared in his case in late March. ... Just 34 years old, he was diagnosed with COVID-induced myocarditis and severe heart failure. Doctors admitted him to the intensive care unit and placed him on a lifesaving intra-aortic balloon pump due to the very poor function of his heart. He spent two weeks in the hospital, has suffered recurrences since his discharge, and now says, “I’m very careful. I’m very concerned about the length of time I’ve been feeling sick, and if these symptoms are lifelong or will go away anytime soon.” J.N. said that everyday activities, like carrying his one-year-old daughter up a flight of stairs, leave him feeling winded and fatigued. He has been unable to work since March

Experts estimate that half of myocarditis cases resolve without a chronic complication, but several studies suggest that COVID-19 patients show signs of the condition months after contracting the virus. One non–peer reviewed study, involving 139 health care workers who developed coronavirus infection and recovered, found that about 10 weeks after their initial symptoms, 37 percent of them were diagnosed with myocarditis or myopericarditis ...

Any such cardiac sequelae lingering weeks to months after the fact is clearly concerning, and we’re seeing more evidence of it. A German study found that 78 percent of recovered COVID-19 patients, the majority of whom had only mild to moderate symptoms, demonstrated cardiac involvement more than two months after their initial diagnoses. Six in 10 were found to have persistent myocardial inflammation. While emphasizing that individual patients need not be nervous, lead investigator Elike Nagel added in an e-mail, “My personal take is that COVID will increase the incidence of heart failure over the next decades.”

Samuel called it “extremely dangerous” for athletes diagnosed with myocarditis to play competitive sports for at least three to six months, because of the risk of serious arrhythmia or sudden death, and several athletes already have made the decision to heed those dire warnings.

And for the rest of us? Wear a mask, social distance, avoid large gatherings, and spend more time in the great outdoors. I would echo the advice of J.N.: “Be careful. Just don’t get the virus in the beginning.” As of today, it’s still the best defense we’ve got.

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Originally posted by JEWELSTAITEFAN:

Quote:

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Originally posted by 1KIKI:
COVID-19 Can Wreck Your Heart, Even if You Haven’t Had Any Symptoms

A growing body of research is raising concerns about the cardiac consequences of the coronavirus


We’ve also known for a while that some COVID-19 patients’ hearts are taking a beating, too—but over the past few weeks, the evidence has strengthened that cardiac damage can happen even among people who have never displayed symptoms of coronavirus infection. And these frightening findings help explain why college and professional sports leagues are proceeding with special caution as they make decisions about whether or not to play.

More than a dozen athletes at Power Five conference schools have been identified as having myocardial injury following coronavirus infection, according to ESPN; two of the conferences—the Big Ten and the Pac-12—already have announced they are postponing all competitive sports until 2021. And in Major League Baseball, Boston Red Sox ace pitcher Eduardo Rodriguez told reporters that he felt “100 years old” as a result of his bout with COVID, and of MLB’s shortened season because of myocarditis—an inflammation of the heart muscle, often triggered by a virus. Said Rodriguez: “That’s [the heart is] the most important part of your body, so when you hear that … I was kind of scared a little. Now that I know what it is, it’s still scary.”

Why are these athletes (and their leagues and conferences) taking such extreme precautions? It’s because of the stakes. Though it often resolves without incident, myocarditis can lead to severe complications such as abnormal heart rhythms, chronic heart failure and even sudden death.

Here’s the background: Myocarditis appears to result from the direct infection of the virus attacking the heart, or possibly as a consequence of the inflammation triggered by the body’s overly aggressive immune response. And it is not age-specific: In The Lancet, doctors recently reported on an 11-year-old child with multisystem inflammatory syndrome (MIS-C)—a rare illness—who died of myocarditis and heart failure. At autopsy, pathologists were able to identify coronavirus particles present in the child’s cardiac tissue, helping to explain the virus’ direct involvement in her death. In fact, researchers are reporting the presence of viral protein in the actual heart muscle, of six deceased patients. Of note is the fact that these patients were documented to have died of lung failure, having had neither clinical signs of heart involvement, nor a prior history of cardiac disease.

Ossama Samuel, associate chief of cardiology at Mount Sinai Beth Israel in New York, told me about a cluster of younger adults developing myocarditis, some of them a month or so after they had recovered from COVID-19. One patient, who developed myocarditis four weeks after believing he had recovered from the virus, responded to a course of steroid treatment only to develop a recurrence in the form of pericarditis (an inflammation of the sac surrounding the heart). A second patient, in her 40s, now has reduced heart function from myocarditis, and a third—an athletic man in his 40s—is experiencing recurring and dangerous ventricular heart rhythms, necessitating that he wear a LifeVest defibrillator for protection. His MRI also demonstrates fibrosis and scarring of his heart muscle, which may be permanent, and he may ultimately require placement of a permanent defibrillator.

J.N. ... told me that COVID-19 symptoms first appeared in his case in late March. ... Just 34 years old, he was diagnosed with COVID-induced myocarditis and severe heart failure. Doctors admitted him to the intensive care unit and placed him on a lifesaving intra-aortic balloon pump due to the very poor function of his heart. He spent two weeks in the hospital, has suffered recurrences since his discharge, and now says, “I’m very careful. I’m very concerned about the length of time I’ve been feeling sick, and if these symptoms are lifelong or will go away anytime soon.” J.N. said that everyday activities, like carrying his one-year-old daughter up a flight of stairs, leave him feeling winded and fatigued. He has been unable to work since March

Experts estimate that half of myocarditis cases resolve without a chronic complication, but several studies suggest that COVID-19 patients show signs of the condition months after contracting the virus. One non–peer reviewed study, involving 139 health care workers who developed coronavirus infection and recovered, found that about 10 weeks after their initial symptoms, 37 percent of them were diagnosed with myocarditis or myopericarditis ...

Any such cardiac sequelae lingering weeks to months after the fact is clearly concerning, and we’re seeing more evidence of it. A German study found that 78 percent of recovered COVID-19 patients, the majority of whom had only mild to moderate symptoms, demonstrated cardiac involvement more than two months after their initial diagnoses. Six in 10 were found to have persistent myocardial inflammation. While emphasizing that individual patients need not be nervous, lead investigator Elike Nagel added in an e-mail, “My personal take is that COVID will increase the incidence of heart failure over the next decades.”

Samuel called it “extremely dangerous” for athletes diagnosed with myocarditis to play competitive sports for at least three to six months, because of the risk of serious arrhythmia or sudden death, and several athletes already have made the decision to heed those dire warnings.

And for the rest of us? Wear a mask, social distance, avoid large gatherings, and spend more time in the great outdoors. I would echo the advice of J.N.: “Be careful. Just don’t get the virus in the beginning.” As of today, it’s still the best defense we’ve got.

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Local Fake News is reporting that Big 10 is planning to open sports this fall, starting next month. With Football Championship on 19 Dec. after 9 weeks of games. Are they ignoring caution, or did they get different info?

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Originally posted by 1KIKI:

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Originally posted by JEWELSTAITEFAN:
Proof that Covid was created in Wuhan Lab?

https://ca.news.yahoo.com/chinese-virologist-claims-she-proof-02570016
4.html

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THANKS for the link, JSF! I saw that in the news and bookmarked it on my 'puter, but - as I don't for probably 95% of my bookmarks - I didn't post it here.

ETA: Despite claims that a laboratory origin has been debunked by this paper https://www.nature.com/articles/s41591-020-0820-9 , the takedown of many such papers due to poor data, faulty analysis, and unjustified conclusions, by a genuine virologist who specializes in viral evolution has left the 'origins' questions open, imo.

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That must be some incredible spike in cases in Portland that you are trying to deny.

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I had thought all reasonable folk had understood that Wuhan Virus was in SoCal before November.

Are you saying you didn't get the news, or are you denying it? Or not infusing your link with known data?

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By sheer numbers... during these 100 days?

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Originally posted by 1KIKI:

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By sheer numbers... during these 100 days?

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What your poor brain seems to be missing is the absence of a SPIKE in Portland related to the protests/(riots).

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Originally posted by JEWELSTAITEFAN:

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Originally posted by 1KIKI:

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Originally posted by JEWELSTAITEFAN:
Proof that Covid was created in Wuhan Lab?

https://ca.news.yahoo.com/chinese-virologist-claims-she-proof-02570016
4.html

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THANKS for the link, JSF! I saw that in the news and bookmarked it on my 'puter, but - as I don't for probably 95% of my bookmarks - I didn't post it here.

ETA: Despite claims that a laboratory origin has been debunked by this paper https://www.nature.com/articles/s41591-020-0820-9 , the takedown of many such papers due to poor data, faulty analysis, and unjustified conclusions, by a genuine virologist who specializes in viral evolution has left the 'origins' questions open, imo.

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Apparently that doc was interviewed on Tucker Carlson a day or so after the story broke, several days sago by now.

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Originally posted by 1KIKI:

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By sheer numbers... during these 100 days?

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What your poor brain seems to be missing is the absence of a SPIKE in Portland related to the protests/(riots).

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Originally posted by 1KIKI:
links to actual data needed

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https://www.portland.gov/omf/covid-19-situation-status-reports-emergen
cy-coordination-center
https://www.portland.gov/sites/default/files/2020-08/2020.08.27_portla
nd-situation-report_final.pdf

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Originally posted by 6IXSTRINGJACK:
Oh... just you wait. There will be plenty of undead voting for Democrats by mail.

I keep telling my Grandpa not to vote for Biden, but I don't think he can hear me through 6 feet of dirt.

Do Right, Be Right. :)

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Originally posted by 1KIKI:

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https://www.portland.gov/omf/covid-19-situation-status-reports-emergen
cy-coordination-center
https://www.portland.gov/sites/default/files/2020-08/2020.08.27_portla
nd-situation-report_final.pdf

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Thanks! for the links.

First of all - those graphs are CUMULATIVE graphs of cases and deaths. OF COURSE they'll continue to go up !! since not too many people will revert to 'never infected' or 'undead'.

If you look at the cases by date that I linked https://multco.us/novel-coronavirus-covid-19/regional-covid-19-data-da
shboard , you'll see that the 'cases' started their rise between the week of May 25, 2020* and May 31, 2020 - far too early to be accounted for by any protests/(riots). There needs to be at least a 2 week lag - for people to become symptomatic and go to the doctor.

And cases then peak the week of July 5, 2020 after which they drop off steeply, far too early compared to the ongoing protests/(riots) which should have sustained the outbreak if they were, indeed, the cause.

Second of all, you need to be careful about increases of 'cases' since - as Fearless Leader has noted - the more tests you do, the more 'cases' you'll find. In this instance, you'll see a disconnect between 'cases' and 'deaths' (after accounting for a 2-3 week lag to give confirmed 'cases' time to become 'deaths'). That indicates more testing, uncovering more 'cases'.

'Deaths' is a better measure (after a 4-6 week lag to confirm 'cases' and for those confirmed 'cases' to become 'deaths'.)

*Third, George Floyd died May 25, 2020. As I noted (above) 'cases' and 'deaths' were occurring far too early to be caused by the protests/(riots).

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CDC abruptly removes guidance about airborne coronavirus transmission, says update 'was posted in error'

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Originally posted by SIGNYM:

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CDC abruptly removes guidance about airborne coronavirus transmission, says update 'was posted in error'

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As I mentioned earlier, I check in with CNN every few days just to keep tabs on the (mostly bullshit) that it spews, and there it was, in big bold headlines, mentioning "covid" and "airborne" in the same sentence! The subhead was that the CDC is recognizing that airborne aerosols are a significant source of transmission. (Gee, whooda thunk? I though the Chinese bus study was pretty telling!) But when I tried to click on the article, several times, all I got was an error.

I guess they couldn't pull the headline fast enough.

The follow-on explanation in ZH was that the WHO put pressure on the CDC to retract the "airborne" designation because ... I dunno. Whatever I read didn't stick because it didn't make sense.

So they are sticking with the 6ft distance, handwashing, and substandard masking which works for large droplets but not aerosols.

It's all a matter of size. The smaller the droplet, the farther it will travel, the longer it will stay suspended in air, and the harder it is to filter out. When someone says "airborne" I mistakenly think of a free-floating naked virus particle, which would be incredibly hard to trap and would probably fall into the category of "ultrafine" particle. But in reality the droplets are probably larger, and more amenable to filtration. As water evaporates the droplets get smaller and smaller, but it look like as the droplets dry out so does the virus, which seems to inactivate it.

Outdoor contagion looks to be rare. So possibly things like bike rallies (Sturgis) and protests (many places) don't result in a massive outbreak.

-----------
Pity would be no more,
If we did not MAKE men poor - William Blake

#WEARAMASK

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Originally posted by JEWELSTAITEFAN:

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Originally posted by JEWELSTAITEFAN:

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Originally posted by 1KIKI:

Quote:

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Originally posted by JEWELSTAITEFAN:
Proof that Covid was created in Wuhan Lab?

https://ca.news.yahoo.com/chinese-virologist-claims-she-proof-02570016
4.html

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THANKS for the link, JSF! I saw that in the news and bookmarked it on my 'puter, but - as I don't for probably 95% of my bookmarks - I didn't post it here.

ETA: Despite claims that a laboratory origin has been debunked by this paper https://www.nature.com/articles/s41591-020-0820-9 , the takedown of many such papers due to poor data, faulty analysis, and unjustified conclusions, by a genuine virologist who specializes in viral evolution has left the 'origins' questions open, imo.

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Apparently that doc was interviewed on Tucker Carlson a day or so after the story broke, several days sago by now.

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I understand there is a 2nd refugee Doctor of Wuhan who is corroborating the information.

He is also reporting that Covid is one of 2 Viruses STORED at the Wuhan facility.

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Originally posted by 1KIKI:
I'd appreciate a name to track this down in all its finer details.

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